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—synaptic neoteny—contributes to ASD. The project will explore how SYNGAP1 haploinsufficiency, a major monogenic cause of ASD, accelerates synaptic development and disrupts early sensory circuit function. A
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neurodevelopmental timing and function. The research will explore: How human-specific genes (SRGAP2C) modulate the trajectory of behavioral maturation Whether accelerated synaptogenesis leads to premature
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