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neuroscience and (4) the development of computational models and methods for neuroscience. The goal is to implement advanced brain-on-chip models and CRISPR-based screens and methodologies to generate
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) and ATTR (transthyretin) amyloidosis are the most common systemic forms, with distinct molecular origins but shared mechanisms of extracellular amyloid deposition and tissue damage. Our research program
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) the development of translational strategies in circuit neuroscience and (4) the development of computational models and methods for neuroscience. The goal is to implement advanced brain-on-chip models and CRISPR
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for Molecular Neurology are recruiting a postdoctoral researcher to establish next-generation humanized and engraftment models for studying peripheral neuroimmune mechanisms driving Parkinson’s disease. Building
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intervention trials (e.g., diet, FMT), and ex vivo gut models enabling advanced multi-omics analyses of these samples. In addition the lab also maintains a large culture collection, partially linked to genomic