46 associate-professor-computer "https:" "https:" "https:" "https:" PhD scholarships at Leibniz
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Pathogens, a WHO Collaborating Centre, and a member of the Leibniz Research Association. The Computational Infection Biology Department, led by Thomas Otto, is seeking a highly motivated PhD Student (in data
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-bips.de by the application deadline. Contact information for further questions Vanessa Didelez, PhD, Program Director, Professor of Statistics with Focus on Causal Inference, didelez(at)leibniz-bips.de
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, computer science, medicine, pharmacology, and physics. ISAS is a member of the Leibniz Association and is publicly funded by the Federal Republic of Germany and its federal states. At our location in
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Leibniz Institute of Plant Biochemistry (IPB) in Halle (Saale), Germany, where we are offering a fully-funded PhD position within the DFG Priority Programme SPP2363: “Molecular Machine Learning”. About the
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, computer science, medicine, pharmacology, and physics. ISAS is a member of the Leibniz Association and is publicly funded by the Federal Republic of Germany and its federal states. At our location in
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part of the Forschungsverbund Berlin (https://www.fv-berlin.de/) and the Leibniz Association (https://www.leibniz-gemeinschaft.de ). You can find more details on the institute webpage: https://www.ikz
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part of the Forschungsverbund Berlin (https://www.fv-berlin.de/ ) and the Leibniz Association (https://www.leibniz-gemeinschaft.de ). You can find more details on the institute webpage: https://www.ikz
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-related traits, and heterosis. In the era of large population size and dense genomic data such as whole-genome sequencing, new algorithms are needed to remove the bottleneck of computational load for such a
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, computer science, medicine, pharmacology, and physics. ISAS is a member of the Leibniz Association and is publicly funded by the Federal Republic of Germany and its federal states. At our location in
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these, Nucleotide-binding, Leucine-rich-Repeat proteins (NLRs) constitute a large family of intracellular receptors of pathogen-associated molecules, termed effectors. To restrict infection, NLRs trigger a response