443 engineering-computation "https:" "https:" "https:" "https:" "UCL" "UCL" "UCL" research jobs at Nature Careers
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innovation, building a profile that combines deep methodological expertise with exposure to real-world technology development. Recent related references: Lesur A, Schmit PO, Bernardin F, et al. Highly
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-of-the-art research platforms provided by the Impresso project : https://impresso-project.ch/ Contribute to teaching and supervision activities at MA level, including seminar preparation and feedback
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this final call. This programme is open to early-career researchers from all disciplines, including Humanities, Social Sciences, Natural Sciences, Health Sciences, and Engineering. Based at the University
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to develop cell-based immunotherapies for pediatric tumors. Studies are focused on using genetic engineering approaches to not only render immune cells cancer specific, but also improve their effector function
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initially for two years with the possibility of extension. We are seeking a highly motivated researcher who is excited to play a key role in developing an ambitious research programme and establishing
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for the game changing impact of our science globally. Our employees enjoy access to state-of-the-art technology and a diverse range of specialist training opportunities, including support for leadership and
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the fields of synthetic biology, immune cell engineering, protein engineering, and immunology, with an eye towards clinical translation. Postdoctoral fellows will gain extensive experience with cutting-edge
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in education, patient care, community service, research and technology development, and through leadership activities in Michigan, nationally and internationally. Our mission is guided by our
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motivated postdoctoral fellow. We study how metabolism and metabolic enzymes in endothelial cells contribute to health and disease by leveraging genetically engineered mouse models, in vitro cell culture with
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Postdoctoral Fellows to join a dynamic, fully funded research program investigating how environmental exposures and inflammation drive hematopoietic stem cell dysfunction, clonal hematopoiesis, and the