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human genetics and disease, toward developing RNA-based therapeutics. • Integrative Analysis of Multi-Omics Data: Build end-to-end workflows for bulk and single-cell RNA-seq, long-read sequencing, and
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BSOM Pharmacology Department Homepage https://pharmacology-toxicology.ecu.edu/ Advertising Department PHARMACOLOGY &TOXICOLOGY Division ECU Health/BSOM Classification Title Research Instructor, Research
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for Development of Gene Therapies TREAT-EXP in the project FENG.02.01-IP.05-M038/25, TREAT-EXP: Development of therapeutic strategies for human genetic diseases caused by nucleotide repeat expansions. The FENG
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scientific discoveries to improve human health locally and around the globe. Composed of more than 2,500 faculty physicians and researchers, more than 1,300 students, and more than 6,000 staff, the Duke
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considered if the position has not yet been filled. Position description Clinical and Translational Researcher http://psych.ucsf.edu/JPF05643 The Department of Psychiatry and Behavioral Sciences
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. no. CZ.10.03.01/00/22_003/0000003) project, Research Program 1 Molecular Biology and Genomics. Workplace Faculty of Science, Department of Biology and Ecology Job description Analyzing genetic structure
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.PEX and DOI identifier https://doi.org/10.54499/2024.15642.PEX ), financed by Fundação para a Ciência e a Tecnologia, I.P. through national funds under the 2024 Call for Exploratory Projects in All
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, gender identity or expression, genetic information or protected veteran status. In accordance with the requirements of Title VII of the Civil Rights Act of 1964, the Title IX of the Education Amendments
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involved in human cohort curation and will conduct independent research by characterizing microbiome composition across multiple anatomical sites using shotgun metagenomics, microbiological and bioinformatic
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University of Massachusetts Medical School | Worcester, Massachusetts | United States | 2 months ago
areas: Developmental nature of vascular anomalies. Our goal is to determine how developmental defects associated with known mutations in humans result in defective vascular or lymphatic function. Studies