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computationally perturbing chiral molecules by smoothly exchanging or adding functional groups or by replacing atoms with those of a different element to understand which changes in molecular structures or solvent
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to such functions as movement, growth and regulation – emerge from interaction between their component parts. A distinctive feature of the CAFE-BIO programme is that a novel combination of modelling and analysis
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key component of the presynaptic boutons, whose function is not limited to storage and release of neurotransmitter. Our previous data indicate that the synaptic vesicle cluster can be seen as a master
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functions. Extrinsically, the ageing tissue environment shows chronic low-grade inflammation, altered cytokine and growth-factor availability, changes in stromal and endothelial cues, shifts in the microbiome
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continuous casting. The process covers 96 % of the total production volume of steel world-wide. The tasks cover the construction of the laboratory demonstrator using low-melting metal alloys including
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the transcription factor LMX1B. The prognosis for patients is determined by damage to podocytes, a cell type essential for the filtration properties of the kidney. The project to be undertaken involves the functional
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PhD project: The genomes of basically all living organisms on our planet are invaded by genetic elements called transposable elements (TEs). These TEs can be considered as parasites in our genome
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scenarios (e.g., metal and welding technology). The findings will be used for direct application and implementation in vocational training curricula and will contribute to improving the design principles
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spectroscopy will be a key component of the experimental toolbox. Where appropriate, additional techniques such as diffraction or photon-based methods and microscopy methods (scanning tunneling microscopy
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crystallography or Cryo-EM, quantitative/biophysical protein interaction studies, spectroscopy and molecular biology. We recently showed that WDR5, a component of the CLOCK/BMAL1 co-activating MLL1 histone