190 software-engineering-model-driven-engineering-phd-position Postdoctoral research jobs at University of Oxford
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data to build hypothesis and test them in laboratory models. You will contribute ideas for new research projects, collaborate in the preparation of scientific reports and journal articles and act as a
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Materials Manufacturing Hub (SCHEMA), and is associated with a project entitled, ‘Hydrogen-driven bio- and chemo-bio-catalysis to unlock diols and carboxylic acids from hydroxymethylfurfural and furfural
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the lead of Prof Daniela Ferreira conducts research using controlled human challenge models to better understand mucosal immunity to respiratory pathogens and inform vaccine development. This position is
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proteome in heart-specific cell lines and primary tissue. It will utilize disease model systems to characterize unique cell surface signatures for cardiomyocytes, coronary endothelial cells, and fibroblasts
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tomato and pepper as model systems. Work in Oxford will build on our extensive experience in studying bacterial virulence mechanisms and the role of the plant microenvironment in disease development
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hold a PhD/DPhil or be near completion of a PhD/DPhil in a relevant biological discipline. Good communication and documentation skills, and the ability to work effectively as part of a team are essential
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induced by radiotherapy. You will employ a broad range of experimental approaches, including cell line engineering, transcriptional reporter assays, flow cytometry, qRT-PCR, ELISA, and CRISPR-based genetic
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approaches including targeted genetic murine models, primary cell culture and analysis, multi-omics and bioinformatics. The biological focus will be on vascular biology, immune cell function and metabolism
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this hydrogen generation model with the ammonia synthesis module. Find out more about the Hayward research and group at: https://www.chem.ox.ac.uk/people/mike-hayward. About you Applicants must hold a
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, activation, and effector functions in preclinical models of autoimmunity. This research is part of a broader effort to define how inhibitory receptors tune T-cell responses in health and disease, ultimately