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of maladaptive innate immune pathways in neuronal stress and death, and (2) examine how mito-nuclear retrograde signaling contributes to microglial dysfunction and senescence in Alzheimerâ™s disease and related
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Department of Biochemistry, in the Temerty Faculty of Medicine at the University of Toronto, invite applications for a full-time tenure stream position in the area of aging, cellular senescence and/or tissue
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of maladaptive innate immune pathways in neuronal stress and death, and (2) examine how mito-nuclear retrograde signaling contributes to microglial dysfunction and senescence in Alzheimer’s disease and related
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of interest include but are not limited to: nutritional approaches to delay cellular senescence, identification of diet-microbe-host interactions relevant to human disease, microbial nutrient metabolism
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Scheduled Hours 40 Position Summary Assists with research study, experiments, preparation of material, and documentation involving cellular senescence, tau biology, and their roles in
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diseases. To do this, they focus on understanding the biology of senescent cells, dysfunctional cells that accumulate in organisms over time and drive aging diseases. They pioneer the development of CAR T
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Sleep Apnea (OSA) contributes to vascular senescence and pulmonary vascular remodeling. This translational research program utilizes a combination of epigenomic, genomic, proteomic, imaging, and
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various transitional senescence states that are generated within the laboratory. Maintains and updates the computational resources assigned to the laboratory within the University’s high-performance
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on funded research project aimed at deciphering the roles of MOAP-1 in cellular senescence and ageing-associated disorders in liver. Long term goal of the project is to gain novel insights towards developing
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Principles of Chromosomal Instability Cellular Senescence, Tumour Suppression and Organismal Ageing Topics covered during the second semester: Materials Sciences Laser Technology Cardiovascular Tissue