14 coding-"https:"-"Prof"-"FEMTO-ST"-"https:"-"https:"-"https:"-"https:"-"https:"-"NOVA.id" positions at City of Hope
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., aberrant DNA methylation, histone modifications, and non-coding RNAs). · Understanding the biological implications of gut microbiome and its translational application in gastrointestinal cancers
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procurement. In depth knowledge of various cancer ontology applications including OncoTree, NACCR and NCCN coding guidelines for anatomic sites, histologic types, staging (T,N,M and overall group) and their
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to various therapies. Study the epigenetic basis of GI cancers (e.g., aberrant DNA methylation, histone modifications, and non-coding RNAs). The genetic basis of early-onset and familial/hereditary colorectal
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with demonstrated analytical capability. Experience with account reconciliation and balancing Knowledge, Skills, Abilities: Solid knowledge of medical terminology, ICD9 codes, CPT/HCPC’s codes. Familiar
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Molecular Dynamics methods and experience in code development is preferred. City of Hope employees pay is based on the following criteria: work experience, qualifications, and work location. City of Hope is
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verifications Knowledge, Skills, Abilities: Knowledge of accounting principles and statistical analyses required Knowledge of medical terminology, ICD10 codes, CPT/HCPC’s codes Advanced written and verbal
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account reconciliation and balancing. Knowledge, Skills, and Abilities: Strong knowledge of medical terminology, ICD-9 codes, CPT/HCPCs codes. Familiarity with Medicare Bad Debt laws and requirements, and
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., aberrant DNA methylation, histone modifications, and non-coding RNAs). · Understanding the biological implications of gut microbiome and its translational application in gastrointestinal cancers
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., aberrant DNA methylation, histone modifications, and non-coding RNAs). · Understanding the biological implications of gut microbiome and its translational application is gastrointestinal cancers
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, and predictive responses to chemotherapies and targeted drugs. 2. Study the genetic and epigenetic basis of GI cancers (e.g., aberrant DNA methylation, histone modifications, and non-coding RNAs). 3