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of Experimental Medicine and Therapy Research, in the laboratory of Dr. Melanie Werner-Klein (https://www.experimentelle-medizin.de/erforschung-der-metastasierung ), we are inviting applications for a highly
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across career stages and genders. Your job Your project will focus on hepatitis viruses, including hepatitis B virus (HBV) and hepatitis C virus (HCV), and their interactions with host cells. The specific
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of fibrosis, accompanied by an inappropriate immune response. It causes approximately 500,000 deaths per year and can progress to other chronic liver diseases, including cancer. Restoring homeostasis in
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(ii) how nutrient‑deprived cancer cells evade death by scavenging nutrients from their environment. We are structural and chemical biologists investigating signaling at biological membranes, with a
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rearrangements, and cell death. Are you passionate about understanding how cells maintain the genome and do you have a desire to use cutting-edge high-throughput methods in combination with the use of fluorescence
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here Project description Cancer remains one of the leading causes of death worldwide, with many types showing resistance to standard treatments, posing significant challenges for healthcare and research
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pneumoniae, a pathogen that causes millions of deaths a year worldwide. Her R01-funded laboratory primarily uses mouse models to study 1) How the actin-bundling protein L-plastin supports immune cell function
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cells and human iPS cells, as well as with genetically modified mouse and pig models. Last publications: https://pubmed.ncbi.nlm.nih.gov/40551151/ https://pubmed.ncbi.nlm.nih.gov/37967560/ https
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Nature Careers | Vancouver South Shaughnessy NW Oakridge NE Kerrisdale SE Arbutus Ridge, British Columbia | Canada | 3 months ago
., Rosberg R., Golec E., Wozniak K., Gomez M.F., Zhang E., O’Connell D., Renström E., and Blom A.M. (2019) Complement C3 is highly expressed in human pancreatic islets and prevents b-cell death via ATG16L1
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to promote the introduction of DNA breaks on the bacterial genome, thereby resulting in genetic instability and cell death if not repaired. Yet despite decades of study, fundamental mechanisms underpinning