279 programming-language "INSAIT The Institute for Computer Science" Postdoctoral positions at Nature Careers
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machine learning methods in the context of biological systems Experience with programming (e.g., Python, Perl, C++, R) Well-developed collaborative skills We offer: The successful candidates will be hosted
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techniques (e.g. explainable, ethical, empathic and agentic AI), natural language processing (NLP), large language models (LLMs), data science methods, and mHealth to analyze large-scale, multidimensional, and
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Ability to quickly adapt to issues by proposing new solutions, open to other researchers’ ideas Team player spirit, and ability to work in a timely fashion to meet deadlines Language skills Fluency in
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., DepMap, PRISM) Common bioinformatics resources and pipelines (e.g., NCBI, Ensembl, BWA, STAR, GATK, VarScan, DESeq) Proficiency in Linux and programming languages such as Python, R, MATLAB, or Perl
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researchers to support complex behavioral experiments. Qualifications: Experience in designing and building custom optical systems. Familiarity with Zemax and CAD software. Strong programming skills in C
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PhD program, you must include a signed letter from your PhD supervisor stating your expected date of completion. Strong collaborative and communication skills and an interest in method development and
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· Excellent written and oral communication skills e.g., in publications, reports, and/or presentations · Collaborative skills, initiative, result oriented, organisation, and capacity to work in a
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-source tools and has a long history in coupling natural language techniques and knowledge graphs. The R&D programme for that position includes several tasks: Generalize and abstract the bot from specific
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, with strong ties to Berlin’s broader biomedical landscape. MDC offers structured postdoctoral training programs, access to cutting-edge technology platforms, and opportunities for interdisciplinary
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prognostic transcriptional programs through an epigenetic switch. Nat Genet. 56(4):663-674. Siersbæk et al., 2020. IL6/STAT3 signaling hijacks ER enhancers to drive breast cancer metastasis. Cancer Cell 38(3