25 phd-in-computational-mechanics-"FEMTO-ST"-"FEMTO-ST" Fellowship positions at City of Hope
Sort by
Refine Your Search
-
development and optimization. The tumor microenvironment and mechanisms of immune evasion. Development of innovative technologies to enhance anti-tumor immune responses. The role of epigenetic and RNA
-
company utilizing GMP manufacturing to produce neoantigen vaccines for patients at medical centers nationally as a primary goal. Your qualifications should include: · PhD degree in an appropriate
-
Metabolism is the interface between cancer treatment and metabolic syndrome. Leveraging multiomics, mouse genetics, hiPS cells, and electrophysiology, the lab works on molecular mechanism of cardiometabolic
-
Postdoctoral Fellow interested in studying the molecular, cellular, and metabolic mechanisms leading to obesity, diabetes, and cancer. The Wang laboratory investigates the function and remodeling of adipose
-
, uncovering basic mechanisms, and identifying novel therapeutic targets in leukemogenesis and solid tumors. Specifically, we study genetic and epigenetic biomarkers with prognostic value in acute myeloid
-
cancer. We seek an independent and highly motivated candidate interested in studying the molecular, cellular, and metabolic mechanisms leading to obesity, diabetes, and cancer. Expertise in using mouse
-
Cancer Journal (2022), Cell Stem Cell (2018). The Li research program is supported by multiple NIH (R01s), American Cancer Society, The Alex’s Lemonade Stand Foundation and several other prestigious
-
computational approaches, to decode tumor and immune biology and usher in a new era of biologically informed clinical care for patients with blood cancer, such as Multiple Myeloma. Our laboratory is part of
-
. Understanding fundamental mechanism of RNA metabolism including splicing and modification is required. Familiar with murine models is preferred. Excellent verbal and written communication skills. Self-motivated
-
. The project involves both in vitro and in vivo studies to uncover the molecular and cellular mechanisms driving cancer-associated immunosuppression using mouse and human solid tumor models. Additionally