24 phd-in-computational-mechanics-"FEMTO-ST"-"FEMTO-ST" PhD positions at University of Cambridge in United Kingdom
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Location: West Cambridge The world-renowned Cavendish Laboratory is seeking an enthusiastic, self-motivated student who enjoys working as part of a team to undertake a PhD in the NanoPhotonics Group
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The Centre for Doctoral Training in Nanoscience and Nanotechnology (NanoDTC) at the University of Cambridge invites applications for its 3.5-year interdisciplinary PhD programme. The programme
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We invite applications from creative and motivated individuals to join Professor Sir Shankar Balasubramanian's group for a 4-year PhD studentship, working on a multidisciplinary project exploiting
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PhD studentship: Defining the role of the pioneer factor FOXA1 in hormone-dependent cancer Supervisor: Professor Jason Carroll Course start date: 1st October 2026 Project details For further
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This is a four-year (1+3 MRes/PhD) studentship funded through the Cambridge EPSRC Centre for Doctoral Training in Future Infrastructure and Built Environment: Unlocking Net Zero (FIBE3 CDT). Further
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Cambridge Institute (CRUK CI), to investigate the molecular mechanisms of small RNA-mediated chromatin silencing. As a whole, the Hannon laboratory has broad interests that range from discovery research
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, Su J, Eldridge MD, Riedel A, Serrao E, Rodewald HR, Mack M, Shields J, Cohen ES, McKenzie ANJ, Goodwin RJA, Brindle K, Marioni JC, Halim TY. (2020) ILC2-driven innate immune checkpoint mechanism
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Primary Supervisor -�Dr Gabrielle Davidson BACKGROUND� Wildlife gut microbiomes are critical for host biology and health, yet are highly variable and environmentally responsive, especially in birds. There�s now a pressing need to understand how these symbionts affect development and fitness....
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details Our lab exploits both chemistry and biology to address fundamental mechanisms of genome function. While many genetic determinants of cancer are known, alternative nucleic acid secondary structures
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treatments that target AR have forced cancer cells to evade treatment and a newly characterised, but frequent escape mechanism is for cancer cells to lose dependence on AR and to take on features of a