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of the tumor suppressor p53, which upregulates the cyclin-dependent kinase (CDK) inhibitor p21. In this project, we aim to investigate the crosstalk between mitotic timing and G1 arrest using engineered cell
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) representation (Research Field “Staging Enmity")and (3) physical encounters (Research Field “Enemy Contact”). Our research regions include Europe, Asia and the Middle East. The RTG Ambivalent Enmity targets
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