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are seeking to appoint a Postdoctoral Research Associate with experience of structural biology techniques to join Dr Paul Elliott’s Lab to investigate the mechanisms of ubiquitin signalling within inflammation
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the molecular mechanisms of ribosome biogenesis dysregulation in cancer. This full-time fixed-term post is funded by Cancer Research UK for 1 year. The actual starting salary offered will be based
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of Rheumatology. This post will be part of a research group with responsibility for carrying out research on mechanisms regulating immunological synapse formation and function. The post holder provides guidance
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with other researchers in designing behavioural tasks and neuroimaging/neurostimulation experiments for investigating the neural mechanisms underlying emotional approach/avoid choices. You will use fMRI
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. The postholders will support ongoing research that aims to unravel the molecular architecture of the chloroplast’s beta-barrel protein assembly machinery using structural tools. One of the posts will be focusing
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personal protection equipment (PPE). Your responsibilities will encompass developing new robotic benchmarking testing setup, hardware and controller of a robotic mechanical impactor, and data acquisition
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with an interest in viral RNA-dependent RNA polymerases and its role in transcribing and replicating the viral RNA genome, as well as on the molecular mechanisms involved in the nuclear import, export
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available data and apply causal inference methods, including Mendelian randomisation, to identify candidate mechanisms linking circadian misalignment and sleep disturbances with cardiometabolic disease
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: Full Time (37.5 hours per week) About the role The Ranes lab are seeking a talented Postdoctoral Research Associate to investigate the molecular mechanisms driving the Wnt/beta-catenin signalling pathway
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mechanisms by which co-inhibitory receptors, including PD-1, BTLA, TIGIT, and CD200R, regulate T cell function in autoimmune settings. Despite sharing common downstream signalling components such as SHP1 and