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lung fibrosis. The ideal candidate will independently perform studies utilizing established in vitro, ex vivo and in vivo preclinical models and will have the opportunity to develop and refine novel
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role areas are: Developing and applying advanced molecular thermodynamic theories and models for water and electrolyte systems relevant to energy, materials, environmental, and biological applications
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Education and qualifications: Required: PhD degree in Biotechnology or related fields. Experience in experimental models and molecular microbiology of serious infections (e.g., bacteraemia, endocarditis
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well as a strong background in immunology, molecular biology, CRISPR-Cas9 gene editing, and flow cytometry for functional assays. Application: Please submit a curricular summary in the FAPESP model , a
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genetic disease modeling, delivery technologies, informatics, and clinical translation of gene therapies are available. Key Responsibilities: Performs a variety of molecular and cell biology experiments and
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. - Conduct high-throughput serum proteomic analyses and integrate molecular datasets. - Validate candidate biomarkers in independent cohorts. WP3.2 – Integrated predictive modeling: - Develop integrative multi
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Department:Biochemistry and Molecular Biology Salary Salary:$38,755.00 - $38,755.00 Position Details Full/Part Time Status:Full Time Percent Time:100% Position Description: The laboratory of Dr. Ksenia Krupina is seeking a
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is on advancing the state-of-the-art in generative modeling and applying these advances toward developing more capable biotechnologies, including design problems that go beyond the molecular scale. We
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combines state-of-the-art computational multiscale modelling (using DFT/TDDFT methods, collision theory, molecular dynamics, stochastic dynamics, Monte Carlo and analytical methods) and its thorough
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The University of North Carolina at Chapel Hill | Chapel Hill, North Carolina | United States | 4 days ago
-tolerance to the insulin producing beta cells, and 2) Development of immunotherapies for T1D. The work makes use of several genetically manipulated mouse models of T1D, as well as humanized mouse models, and