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studying the disruption of calcium homeostasis in the setting of cardiac and neuronal damage. Studies underlying molecular signaling. Utilizes approaches including the extensive use of mouse models
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on the role of epigenetic dysregulation for future clinical applications, particularly on their common Mantle cell lymphoma (MCL) model, through their French networks (LYSA, Carnot Institute CALYM) and European
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relevant large animal model of cerebral vascular disease and blood-brain barrier disruption caused by epileptic seizures and hypoxia-asphyxia. Placing particular emphasis on endogenous antioxidant
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experiments using magnetogenetics to selectively control neurons and astrocytes in Alzheimer’s disease models, in vitro and in vivo . Perform in vivo two-photon imaging, electrophysiology, and behavioral tests
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The Max Delbrück Center for Molecular Medicine in the Helmholtz Association aims to transform tomorrow’s medicine through our discoveries of today. At locations in Berlin-Buch, Berlin-Mitte
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metrics and benchmarking pipelines for network inference methods. Demonstrate how these models uncover biological principles and insights across molecular, cellular, and population levels. Build open-source
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, biomechanics, single cell-analyses, advanced imaging, and animal models. Tasks: to actively pursue the proposed project with the aim of characterizing the molecular, biomechanical and functional mechanisms
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Schizosaccharomyces pombe as a model organism. Representative publications include: Nature (2011), PMID: 21725325; Molecular Cell (2016), PMID: 27666591; Cell Reports (2016), PMID: 26832414; PNAS (2021), PMID
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Biochemistry, Molecular/Cell Biology, and/or BioEngineering documented by peer-reviewed publications Skills in a broad range of methods in the fields of Biochemistry, Molecular/Cell Biology, and/or
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heart disease as well as other medical conditions. We are determining single-cell based molecular mechanisms that are disrupted in mouse models of 22q11.2 deletion syndrome in early staged mouse embryos