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their area of interest and will serve as their primary supervisor. The student will become part of the faculty member’s research group, which typically includes master’s students, PhD students, postdocs, and
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-cell RNAseq datasets and analysis tools, large high-dimensional flow cytometry phenotyping panels and a collection of transgenic mouse models specifically engineered for the reporting, lineage tracing
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as well as in aging mouse models. Identification of these interactions could provide new approaches to counteract or delay age associated exhaustion of HSC. PhD Project: Contribution of cellular
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experience growing, managing, and phenotyping plants in the field and greenhouse. You have experience using modern approaches in root phenotyping, image analysis, or simulation modeling to understand
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combining: Molecular synthesis Electrical contacting of nanoscale electronics Modelling of properties on an atomistic level The IHRS NanoNet offers an international scientific environment and an excellent
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strives to offer a best-practice model of doctoral education. This includes a balanced curriculum, innovative teaching methods and the personal engagement of all involved advisers, international cooperative
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an emphasis on in vivo assays, animal models and state-of-the-art imaging tools Soft skills seminars on communication, presentation and other topics equally relevant for achieving success in both a scientific
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aging in Cockayne syndrome (CS) patients. Chromatin serves as a platform for DNA repair and undergoes dynamic changes during the DNA damage response (DDR) through the Access-Repair-Restore model. Histones
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Max Planck Institute for Demographic Research (MPIDR) | Rostock, Mecklenburg Vorpommern | Germany | about 7 hours ago
-motivated and qualified candidates to work with an international team on developing cutting-edge novel demographic, statistical and computational methods in estimating, modelling and forecasting measures
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the Access-Repair-Restore model. Histones, an essential component of chromatin, are post-translationally modified via methylation, ubiquitination, and acetylation to regulate DDR-related chromatin functions