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human health trajectories through advanced digital twin frameworks. This position offers an extraordinary opportunity in the field of computational health modeling that integrates clinical, genotypic
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, enthusiastic, and collaborative candidates. We employ both basic sciences and engineering approaches to modulate inflammatory responses after neural trauma. Currently NIH-funded projects for postdoctoral
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releases tissue factor positive microvesicles that trigger coagulation and drive pathology in infection and sterile inflammation using in vivo models. Our recent work uncovered NINJ1, a key mediator of
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models (e.g., iPSC, organoids, microfluidics) Bioinformatics, ‘omics, big data, molecular genomics/CRISPR/epigenomics Behavioral science, biobehavioral, psychosocial, translational, implementation science
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. Experience or willingness to work with mouse models is required. Proficiency in mouse experimentation, cell culture, biochemical and molecular biology techniques, and immunohistochemistry is highly desirable
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spectroscopic observations with theoretical models and synthetic advances, contributing to the broader effort of designing next-generation quantum and optoelectronic materials. Ideal candidates will have a strong
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releases tissue factor positive microvesicles that trigger coagulation and drive pathology in infection and sterile inflammation using in vivo models. Our recent work uncovered NINJ1, a key mediator of
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inter‐organ communication in heart injury (e.g. heart failure) and neurological disorder (e.g. traumatic brain injury) using our unique genetic mouse models. Candidates with research experience in
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small animal models is also desirable. The ideal candidate will be highly independent but also able to work collaboratively to advance the research projects collectively. Strong written and verbal
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small animal models is also desirable. The ideal candidate will be highly independent but also able to work collaboratively to advance the research projects collectively. Strong written and verbal