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. The postholders will support ongoing research that aims to unravel the molecular architecture of the chloroplast’s beta-barrel protein assembly machinery using structural tools. One of the posts will be focusing
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This project focuses on understanding the molecular mechanisms by which co-inhibitory receptors, including PD-1, BTLA, TIGIT, and CD200R, regulate T cell function in autoimmune settings. Despite sharing common
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mechanisms by which co-inhibitory receptors, including PD-1, BTLA, TIGIT, and CD200R, regulate T cell function in autoimmune settings. Despite sharing common downstream signalling components such as SHP1 and
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of Spiralian Asymmetric Cell Divisions”. This research position will reveal the mechanisms that drive the evolution of polar lobes during the first asymmetric cell divisions in animals with spiral cleavage. We
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collaborative and internationally renowned research environment. The successful candidate will play a key role in advancing cutting-edge research focused on understanding the physiological mechanisms underlying
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on the molecular mechanism of DNA replication initiation in bacteria. This work is aimed at unravelling the sequence of events that lead to helicase loading during initiation and restart. This structural biology
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explore the mechanism by which Wnt signalling can promote successful cytokinesis in the early C. elegans embryo. Cytokinesis, the division of one cell into two, is crucial for an organism’s development and
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. Ready to be part of our team? Let’s shape the future together! About the team: Our group develops and applies quantum mechanics-based methods for the description of condensed matter systems, including
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cellular interactions in tissue, better understand and model the mechanisms driving inflammation and aging, and capture spatio-temporal dynamics in development, all with the aim of uncovering new biology
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of pathogenicity genes between microbes and can therefore drive the emergence of novel diseases. In pathogenic fungi, an emerging global threat to both humans and our crops, the mechanisms that facilitate HGT remain