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applicant will have strong background in SKIN RESEARCH, wound healing, fibrosis or inflammatory disorders with technical expertise in induced pluripotent stem cells (iPSCs), immunology, cell biology or signal
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, advanced imaging, animal models, and biomaterials to develop new strategies to modulate wound healing and fibrosis. This person will be performing semi-technical laboratory and research procedures under
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translate this understanding into disease alleviation and prevention. For more details, please view https://www.ntu.edu.sg/medicine/research/research-programmes/nutrition-metabolism-health We are seeking a
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University of California, San Francisco | San Francisco, California | United States | about 1 month ago
. The salary range for this position is $68.57 - $85.76 (Hourly Rate). To learn more about the benefits of working at UCSF, including total compensation, please visit: https://ucnet.universityofcalifornia.edu
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conferences. Curious to learn more? Explore the full role description below to discover more about this opportunity. The team The ReXIL and Cystic Fibrosis Airway Research Groups are a team based in Respiratory
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Cardiovascular Research Inst Full Time 88032BR Job Summary The Airway Clinical Research Center (ACRC) integrates research programs in asthma, COPD, sarcoidosis, and cystic fibrosis, along with
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Social Worker - Cystic Fibrosis Clinic. WORK SCHEDULE Part-Time / 32 hours per week Day Shifts (Monday/Thursday on-site and 2 day telework) This position is expected to last through 6/30/2026 to be renewed
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, interstitial lung disease, pulmonary hypertension, bronchopulmonary dysplasia, cystic fibrosis, sleep medicine, the pediatric airway and medical education. Our section has a significant research portfolio
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Compensation Plan. UC Salary information can be found here: https://www.ucop.edu/academic-personnel-programs/compensation/2024-25-academic-salary-scales.html . Application Window Open date: May 17, 2025 Most
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Summary: Our lab aims to understand the mechanisms underlying metabolic remodeling and cardiac fibrosis in hypertrophic cardiomyopathy (HCM), using mouse models and human induced pluripotent stem cells