40 computational-solid-mechanics Postdoctoral positions at Baylor College of Medicine
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@bcm.edu". Please include "apply for a postdoc in bioinformatics" in your email subject. Job Duties Develops and/or applies computational approaches to understand the mechanism of cancer development
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on understanding immune mechanisms underlying resistance and relapse in hematologic malignancies, with a goal of translating discoveries into clinical applications. We are part of a vibrant cancer immunology and
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a combination of biochemical techniques, X-ray crystallography, cryoEM, and cryoET techniques, focusing on mechanisms underlying the virus entry, antibody neutralization, replication, morphogenesis
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Neurological Research Institute, Texas Children’s Hospital. Immediate position available at the level of Postdoctoral Associate to study the mechanisms underlying astrocyte-tumor interactions. The focus
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research team focused on dissecting the immune microenvironment in acute myeloid leukemia and post-allogeneic hematopoietic cell transplantation. Our work investigates mechanisms of T cell dysfunction
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Biology in Cancer Genomics and Immunology. This position will be involved in the development and/or application of computational approaches to understand the mechanism of cancer development, progression
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groups pioneered human organoid models to study host-virus interactions, and ongoing work is expanding these systems to include microbial communities. The ultimate goal is to uncover mechanisms and
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to join our dynamic research team investigating post-transcriptional and epigenetic mechanisms that control cell fate, stem cell plasticity, and cancer progression. Research Focus We employ cutting
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devoted to unraveling the mechanisms of how central nervous system orchestrates systemic energy balance, aiming to unlock groundbreaking therapies for obesity and metabolic disorders. The research projects
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the molecular mechanisms driving therapy resistance in human cancers. The research project focuses on understanding the molecular mechanisms by which MAPK4 and/or COP1 regulate cancer progression and therapy