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Field
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computational collaborators to explore drug resistance and immune evasion mechanisms in leukemia, such as clonal evolution, cancer antigen presentation, and cancer-immune crosstalk. Additionally, the fellow will
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underlying sleep, a fundamental and evolutionary conserved behavior. We are studying the homeostatic and circadian mechanisms regulating sleep, and also have deep interest in understanding the functions
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will join a multidisciplinary research program that combines experimental models, patient-derived materials, and advanced technologies to explore the mechanisms that preserve auditory system homeostasis
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focuses on mechanisms of regulation of the innate immune system and how defects in these processes impact immune-mediated diseases such as Inflammatory Bowel Disease (IBD) and systemic autoimmunity
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partners and will innovate new mechanisms to secure future computing systems at lower costs and performance overheads. About you You should possess a PhD/DPhil in Engineering, Computer Science or other
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Oxford University Weighting of £1,500 per year (pro rata). Are you passionate about taking a lead role in a cutting-edge project at the intersection of genomics, computational biology, and haematological
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advanced computational methods to analyze and integrate single-cell multiomics datasets to decipher cellular and molecular mechanisms for disease heterogeneity. · Actively identify new opportunities
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detectors while also having flexibility to pursue your own research interests. Research Focus Participate in a detector R&D program aimed at developing superconducting nanowire sensors to enable
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curious to deliver work that matters, your journey starts here! Innovation. Interdisciplinary collaboration. Complex problem solving. In Carnegie Mellon University’s Department of Mechanical Engineering
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of uncovering micro- and nanoscale deformation mechanisms of cells within their native tissue environments, under both healthy and pathological conditions (e.g., osteoarthritis, inflammation).Experimental data