100 computational-cell-signaling-post-doc Postdoctoral positions at University of Washington
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projects include 1) defining the mechanism of genetic factors influencing pulmonary arterial hypertension, 2) evaluating how endothelial cells crosstalk with other lung cell populations in the disease lungs
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Position Summary The Foltz lab works at the intersection of translational immunology and computational biology. We study mechanisms of response and resistance to natural killer (NK) cell therapies
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, particularly in the areas of the actin cytoskeleton and membrane trafficking. • Utilize quantitative, biophysical, and computational approaches to cell biology, including fluorescence imaging, image analysis
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Details Posted: Unknown Location: Salary: Summary: Summary here. Details Posted: 24-Aug-25 Location: Seattle, Washington Type: Full Time Categories: Academic/Faculty Internal Number: 142057 Position
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from the ovarian stroma and transform these signals into biochemical signals within the follicle (See Amargant et al., 2020, Aging Cell; Pietroforte et al., 2023 Under review PNAS). 2. How does oocyte
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Details Posted: Unknown Location: Salary: Summary: Summary here. Details Posted: 24-Aug-25 Location: Seattle, WA Categories: Academic/Faculty Internal Number: 148986 The postdoctoral scholar would
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. Computational and bioinformatic skills. Experience in microscopy. Generation and analysis of mouse models. Handling of human samples. Molecular biology skills including CRISPR, cloning and qPCR. In vitro cell
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, creative, independent, and enthusiastic scientist to further our basic and translational studies of inflammatory bowel disease (IBD). We study disease-relevant cell and organ signaling pathways in IBD
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pulmonary vascular endothelial cells contributes to pulmonary vascular remodeling. Our current research program includes investigations into the role of hyperactive mTOR (mechanistic target of rapamycin) in
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biochemistry, genomics, molecular genetics, cell biology, and model organism systems to uncover the mechanisms by which histone mutations disrupt human development and lead to disease. Our ultimate goal is to