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Full time, Fixed Term (up to 20 months) Closing date: 23:59 30 September 2025 Interview date: 10 October 2026 (with the expectation that the appointment will start as soon as possible thereafter). The PDRA in Functional Biodiversity will work on the LEMONTREE (Land Ecosystem Models based On New...
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ABOUT THE ROLE Northumbria University invites applications for a Post-Doctoral Researcher to join the EVIDENT project (Evidence-Based Architectural Design of Emergency Departments for Greater
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United Kingdom Application Deadline 14 Sep 2025 - 00:00 (UTC) Type of Contract Other Job Status Full-time Is the job funded through the EU Research Framework Programme? Not funded by a EU programme Is the Job
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movement through the soil-root-stem-leaf-atmosphere system and reveal those traits at each point in the system that contribute to enhanced yield stability in maize. The successful candidate will work in
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potential emissions reduction and improvement in soil health. The post will rely strongly on project collaboration with local NGO and government stakeholders as well as the multiple university-based research
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of Reading, Sheffield and Newcastle, the Met Office, Environment Agency, National Energy System Operator and the Wildlife Trust. Here we have an exciting opportunity for a Post Doctoral Research Associate
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the processes of water movement through the soil-root-stem-leaf-atmosphere system and reveal those traits at each point in the system that contribute to enhanced yield stability in maize. The successful candidate
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slavery and war. Structured around four interconnected research strands—(Re)conceptualising, Understanding, Forecasting and Tackling—the Centre’s programme aims for far-reaching insights that transform
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these pathways modulate T-cell signalling, activation, and effector functions in preclinical models of autoimmunity. This research is part of a broader effort to define how inhibitory receptors tune T-cell
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mechanisms by which co-inhibitory receptors, including PD-1, BTLA, TIGIT, and CD200R, regulate T cell function in autoimmune settings. Despite sharing common downstream signalling components such as SHP1 and