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concretely your work package, for the preparation of a doctorate, contains: Are you a bioinformatician interested in how genetic differences influence cell development? Join us as a PhD candidate at the
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data. We have developed in vivo single-cell CRISPR technologies to screen for dozens of molecular factors in vivo during developmental and disease. These technologies are a game-changer in the speed
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mimicked with in vivo models of metastasis, which provides unique opportunities to mechanistically dissect what drives the different cell states. You will link clinically relevant phenotypes to putative
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genetics, cell biology, genomics, and bio-computing to unravel plant biological processes and to further translate this knowledge into value for society. Please visit us at www.psb.ugent.be for more
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. Clinical studies have demonstrated that hereditary neuropathies can also involve muscle pathologies, complicate diagnosis and hamper therapy development. We have developed advanced in vitro cell models
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. Clinical studies have demonstrated that hereditary neuropathies can also involve muscle pathologies, complicate diagnosis and hamper therapy development. We have developed advanced in vitro cell models
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mechanisms involving genetic, epigenetic and chromatin organisation factors governing the cell-type specific response to ARS mutations. Therefore, integrative genome-wide cis-regulatory network analysis
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neuroinflammation. The PhD student will help uncover the molecular and cellular pathways linking the allergic gut to the inflamed brain, and how these pathways shape disease risk later in life. The successful
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team of Prof. Seppe De Schepper investigates how peripheral immune signals influence brain health and disease. We focus on tissue-resident macrophages and immune cell trafficking from periphery to brain
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well as the available public data suggest mechanisms involving genetic, epigenetic and chromatin organisation factors governing the cell-type specific response to ARS mutations. Therefore, an integrative genome-wide cis