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biology. The project aims to uncover how mechanical properties, forces, and physical phenotypes integrate with molecular networks to regulate the function of complex cellular systems across multiple
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. Responsibilities include but are not limited to the following areas: Conduct cutting-edge research to elucidate cellular and molecular mechanisms underlying stem cell regulation in the context of regeneration, aging
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The International Institute of Molecular Mechanisms and Machines Polish Academy of Sciences | Poland | about 2 hours ago
exploit cellular translation” Download the description of the project: https://projekty.ncn.gov.pl/opisy/566264-en.pdf Short description of the project: We are interested in studying how bunyaviruses
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include identifying relevant molecular targets, linking cellular mechanisms and defects to disease phenotypes, and small molecule screening to identify therapeutics. Understanding vascular mural cell
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development of blood and prostate cancers. Our research bridges basic, translational, and clinical studies to uncover mechanisms linking inflammation, DNA damage, and stem cell aging to cancer initiation and
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Line of research: Mechanisms of nuclear envelope remodelling during mitosis and their role in micronuclei formation Foreseen start date: February 2026 (Flexibility) Professional category
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Postdoctoral Researchers to study the molecular mechanisms linking DNA replication, chromatin dynamics, and genome stability in cancer. Scientific Background DNA replication is essential for cellular
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development of blood malignancies and other solid cancers. Our research bridges basic, translational, and clinical studies to uncover mechanisms linking inflammation, DNA damage, and stem cell aging to cancer
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cell-derived muscle models or 3D culture systems (including physiological hypoxia) is a plus. Experience in biochemical and immunological mechanisms of immune-suppression Position Type Research
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through migration and morphogens release in long range-patterning of cortical regions. However, their developmental origins and the molecular mechanisms underlying CR function in humans remain poorly