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will also have experience dissecting signaling pathways of cell growth, differentiation, senescence and/or death. Skilled in molecular biology, mammalian cell culture, confocal imaging, bioinformatics
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. - Proven experience in safety assays using cell cultures and other models, namely in assessing cell proliferation/cell death. - Ability to analyse experimental results, with solid knowledge in statistical
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of maladaptive innate immune pathways in neuronal stress and death, and (2) examine how mito-nuclear retrograde signaling contributes to microglial dysfunction and senescence in Alzheimerâ™s disease and related
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death. The role of NLR genes in these processes has been thoroughly described for plants and animals, but how they contribute to the fungal immune system is unexplored. Fungal NLR genes also show hugely
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of maladaptive innate immune pathways in neuronal stress and death, and (2) examine how mito-nuclear retrograde signaling contributes to microglial dysfunction and senescence in Alzheimer’s disease and related
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officers may access service rifles when there is an in-progress threat with an individual brandishing a firearm, an active shooter, or an individual actively committing serious bodily injury or death with a
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: https://s.ua.es/es/nlJx , approved by the Governing Council of 22 December 2009 (BOUA of 15 January 2010), and with instructions of the University of Alicante Offices of the Registrar and the Comptroller
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Investigating Roles for C9orf72 in Regulating FIS1-mediated Cell Death and Survival School of Biosciences PhD Research Project Self Funded Dr C Guo, Dr Kurt de Vos Application Deadline
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Hirszfeld Institute of Immunology and Experimental Therapy Polish Academy of Sciences | Poland | 3 months ago
of various synthetic and natural substances, peptides, and antibodies. Investigation of signalling pathways leading to cell death is required. Laboratory experience in eukaryotic cell culture and functional
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: connexin dysfunction in astrocytes, a type of support cell in the brain and spinal cord. Astrocytes play a vital role in maintaining motor neuron health, but when they malfunction, they can become toxic and