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one of the drivers of cellular transformation and cancer development. Tumor suppressor p53 prevents proliferation of cells with damaged DNA and eliminates them either by programmed cell death or by
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management. Cell culture techniques: maintenance of cell lines, cell proliferation and death assays, infections and transfections. Molecular biology techniques: western blot, RT-qPCR, flow cytometry
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-immunology-and-cancer-cell-death Where to apply Website https://www.jobbnorge.no/en/available-jobs/job/291171/researcher-in-cancer-immu… Requirements Research FieldMedical sciencesEducation LevelPhD
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for patients resistant to current treatments The candidate will join Team 11 CRCI2NA and benefit from established expertise in cell death, epigenetics, omics, and technical support of the team. Where to apply E
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cells and human iPS cells, as well as with genetically modified mouse and pig models. Last publications: https://pubmed.ncbi.nlm.nih.gov/40551151/ https://pubmed.ncbi.nlm.nih.gov/37967560/ https
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the University of Houston. The lab employs molecular, cellular, electrophysiological and biochemical approaches to investigate the mechanism of photoreceptor cell death and the application of gene therapy in mice
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, functional, and phenotypic datasets. Oversee analysis and interpretation of large-scale bioinformatic datasets in collaboration with computational researchers. Investigate cell fate and cell death pathways as
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of Biomedical Sciences, Faculty of Health and Medical Sciences, University of Copenhagen, to commence on 01.04.26. Information on the department can be found at: https://bmi.ku.dk/english/about_department
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on large-scale sequencing data or murine models. Primary cells will be incorporated into a commercial ToC model (idenTx3), stained with apoptosis and cell death dyes, and imaged using a live-imaging
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Nature Careers | Vancouver South Shaughnessy NW Oakridge NE Kerrisdale SE Arbutus Ridge, British Columbia | Canada | 20 days ago
., Rosberg R., Golec E., Wozniak K., Gomez M.F., Zhang E., O’Connell D., Renström E., and Blom A.M. (2019) Complement C3 is highly expressed in human pancreatic islets and prevents b-cell death via ATG16L1