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Carnegie Mellon University is a private, global research university that stands among the world’s most renowned education institutions. With ground-breaking brain science, path-breaking performances
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neuroscience, with the aim of improving mental health outcomes in both non-clinical and clinical populations. We are based at the MRC Cognition and Brain Sciences Unit, University of Cambridge, a world-leading
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, beginning July 1, 2024 by the Alliance for Therapies in Neuroscience (ATN), a research partnership between academia and industry to transform the fight against brain diseases and disorders of the central
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to lead impactful research at the interface of aging biology, neurodegeneration, and spatial omics. The successful candidate will contribute to high-profile projects investigating the cellular and molecular
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University of North Carolina at Chapel Hill | Chapel Hill, North Carolina | United States | 1 day ago
disrupted by neurological disorders and injuries. The lab uses high-density neurophysiology (Neuropixels), high-density spinal stimulation, brain-spine interfaces, computational modeling, computational
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with an international resonance as well as to offer teaching and talent development of the highest quality. The school has a broad cooperative interface with society as a whole, both in Denmark and
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are seeking a motivated and enthusiastic colleague with strong computational skills in the analyses of complex data sets to join our teams. About the project We have generated advanced brain on chip models
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are seeking a motivated and enthusiastic colleague with strong computational skills in the analyses of complex data sets to join our teams. About the project We have generated advanced brain on chip models
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at the interface of experimental and computational system neuroscience. Responsibilities Responsibilities include: Conducting research experiments in the field of neuroscience under a research scope and
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microscopy of radial glial cells, RNA sequencing on the developing cortex and CSF metabolomics to understand how microbial signals at the CSF could modulate RG at the ventricular interface. Experience in mouse