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University of North Carolina at Chapel Hill | Chapel Hill, North Carolina | United States | about 19 hours ago
the NIH-funded Emory-Sage-SGC-Jax TREAT-AD Center. This multi-institutional initiative is focused on accelerating the discovery of new therapeutic targets and strategies for Alzheimer’s disease by
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will design climate change mitigation scenarios that respect both climate targets and sustainable development goals using the REMIND model, one of the world’s leading integrated energy-economy-climate
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evidencing: which scientific discoveries are more impactful than others; whether public attitudes to science change over time; how the public learn and talk about science; how different target groups respond
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challenging research and work in a highly collaborative manner. Projects will discover potential new targets to guide the personalized treatment of devastating human cancers of unmet need. For more information
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and PhD students. Research spans a wide range. Current interests include: Bayesian statistics; modelling of structure, geometry, and shape; statistical machine learning; computational statistics; high
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-targeting motifs. Design and synthesize chloride-sensitive fluorescent indicators and organelle-targeting motifs. Create organelle-selective imaging tools for organelle chemotype fingerprinting, a key aim of
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proteins with regard to their role in the development of cancer, the emergence of therapy resistance, as predictive markers to guide therapy and as therapeutic targets. Your Responsibilities Independent
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-pathogenic fungi and identifies targets for the development of novel natural product-based antibiotics. The junior research group Metabolomics-guided Natural Product Discovery (https://www.leibniz-hki.de/de
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and their microenvironment, with a focus on immune cells, stromal components, and their metabolic communication. One central aim is to identify novel therapeu-tic targets for next-generation immune and
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arthritis (RA). The projects will build upon recent publications demonstrating how a unique bacterium targeted by autoantibodies from individuals with RA can stimulate the development of autoimmunity and